Irrefutable: The coronavirus was engineered by scientists in a lab using well documented genetic engineering vectors that leave behind a “fingerprint”
Feb 3, 2020 | Opinion
(Natural News) Every virology lab in the world that has run a genomic analysis of the coronavirus now knows that the coronavirus was engineered by human scientists. The proof is in the virus itself: The tools for genetic insertion are still present as remnants in the genetic code. Since these unique gene sequences don’t occur by random chance, they’re proof that this virus was engineered by scientists in a lab.
But the WHO and CDC are covering up this inconvenient fact in order to protect communist China and its biological weapons program, since no government wants the public to know the full truth about how frequently government-run labs experience outbreaks. Decades ago, for example, the U.S. Army ran an Ebola bioweapons lab in the United States, where a monkey infected one of the scientists there. The strain turned out to be infectious only in monkeys, not humans, so the world dodged a bullet, but the U.S. Army “nuked” the entire facility with chemical bombs, killing all the monkeys and wiping out any last remnant of the virus on U.S. soil.
You can read the full details of that incident in the book The Hot Zone by Richard Preston. We’ve also covered it at NaturalNews.com, where this book description is reprinted:
In 1989, Reston, VA — one of the most famous U.S. planned communities located about 10 miles from Washington DC — stood at the epicenter of a potential biological disaster. This well-known story was narrated by Richard Preston in a bone chilling account related to the recognition and containment of a devastating tropical filovirus at a monkey facility — the Reston Primate Quarantine Unit.
That outbreak occurred because Ebola was found to be spreading through the air ducts, confirming that Ebola can spread through the air. This simple fact was vigorously covered up by the entire medical establishment during the Ebola scare in the United States many years later, where the CDC transported an infected patient to a hospital in Dallas, subsequently infecting a nurse who was treated with highly toxic chemicals that caused permanent kidney damage (she later sued the hospital for the damage she suffered).
The reason this is relevant is because in order to understand the coronavirus situation in China, we must first realize that virology research labs routinely experience lapses in containment. Even the United States has failed to contain deadly viral strains when trying to study them. China’s BSL-4 labs have experienced multiple accidental releases of SARS strains, and this new coronavirus is now confirmed to be an engineered strain that was either used in bioweapons research or vaccine experiments.
The genomic coding in the virus is not natural, in other words. Just as you would never encounter a snake in the desert that’s writing a book containing words and grammatical structure, the genetic sequences now identified in the coronavirus strain are, without question, proof that human engineers have been tinkering with the strain.
How to genetically engineer viruses: the pShuttle vector
One of the tools used to accomplish this genetic engineering is called pShuttle. It’s a genetic tool set that can carry a payload of genes to be inserted into the target virus.
Researchers engaged in genetic engineering can purchase the pShuttle sequence from online retailers such as AddGenes.org, which sells the sequence for $75, shipped in “bacteria as agar stab.”
The following map outlines the complete gene sequence of the pShuttle tool:
The method for using pShuttle is described in a PubMed document entitled, “A simplified system for generating recombinant adenoviruses.”
The summary of the paper describes, “a strategy that simplifies the generation and production of such viruses.” Here’s how the process works to achieve genetic engineering of viruses:
A recombinant adenoviral plasmid is generated with a minimum of enzymatic manipulations, using homologous recombination in bacteria rather than in eukaryotic cells. After transfections of such plasmids into a mammalian packaging cell line, viral production is conveniently followed with the aid of green fluorescent protein, encoded by a gene incorporated into the viral backbone. Homogeneous viruses can be obtained from this procedure without plaque purification.
The paper describes how this approach will, “expedite the process of generating and testing recombinant adenoviruses.”
During this process, of course, the pShuttle leaves behind unique code, a “fingerprint” of the genetic modification. It is this fingerprint that has now been identified in the coronavirus.
As revealed by genomics researcher James Lyons-Weiler in this bombshell analysis article, the pShuttle genetic code is found in the coronavirus that’s circulating in the wild.
This is proof that the virus has been engineered by human scientists.
“IPAK researchers found a sequence similarity between a pShuttle-SN recombination vector sequence and INS1378,” writes Lyons-Weiler for IPAK:
Another gene sequence also shows a 92% match with the Spike protein from the SARS coronavirus:
The process for achieving this was patented by Chinese researchers as shown in this patent link.
The pShuttle vector was used to insert SARS genes into the coronavirus, a process that makes it deadly to humans. “The very researchers conducting studies on SARS vaccines have cautioned repeatedly against human trials,” warns Lyons-Weiler:
The disease progression in of 2019-nCoV is consistent with those seen in animals and humans vaccinated against SARS and then challenged with re-infection. Thus, the hypothesis that 2019-nCoV is an experimental vaccine type must be seriously considered.
He also warns about, “studies that have reported serious immunopathology in animals – rats, ferrets, and monkeys – in which animals vaccinated against coronoviruses tended to have extremely high rates of respiratory failure upon subsequent exposure in the study when challenged with the wild-type coronavirus.”
If the Chinese government has been conducting human trials against SARS. MERS, or other coronviruses using recombined viruses, they may have made their citizens far more susceptible to acute respiratory distress syndrome upon infection with 2019-nCoV coronavirus.
Wuhan Coronavirus, MRC Centre for Global Infectious Disease Analysis
Summary Report 5
Genetic diversity of SARS-CoV-2 (formerly 2019-nCoV), the virus which causes COVID-19, provides information about epidemic origins and the rate of epidemic growth. By analysing 53 SARS-CoV-2 whole genome sequences collected up to February 3, 2020, we find a strong association between the time of sample collection and accumulation of genetic diversity. Bayesian and maximum likelihood phylogenetic methods indicate that the virus was introduced into the human population in early December and has an epidemic doubling time of approximately seven days. Phylodynamic modelling provides an estimate of epidemic size through time. Precise estimates of epidemic size are not possible with current genetic data, but our analyses indicate evidence of substantial heterogeneity in the number of secondary infections caused by each case, as indicated by a high level of over-dispersion in the reproduction number. Larger numbers of more systematically sampled sequences – particularly from across China – will allow phylogenetic estimates of epidemic size and growth rate to be substantially refined.
As of 1500 GMT 14 February 2020, almost 64,000 cases of COVID-19 (formerly 2019-nCoV) have been reported in China (with 1381 deaths), and cases have been detected in at least 24 regions or countries outside mainland China. Since the identification of the novel coronavirus disease from Wuhan City, China in December 2019 there have been ongoing efforts to identify the source of the virus. Many of the cases reported at the beginning of the epidemic had an epidemiological link to a seafood and wildlife market in Wuhan City pointing to a single spillover event from an animal reservoir . Initial phylogenetic analysis showed that the new virus was similar to the SARS coronavirus when compared with other coronaviruses known to infect humans, with bats or pangolins as the most likely animal reservoir [2–4].
Genetic data can give insights into the evolving epidemic which are not always apparent from the epidemiological data alone. As a virus is transmitted through a population, it will accumulate mutations in its genetic code. By identifying changes in genetic sequences sampled from different patients, it is possible to reconstruct the evolutionary history of an epidemic. For a novel pathogen of zoonotic origin this can allow us to estimate when the pathogen was introduced into the human population. Population genetic modelling can also be used to infer population size trends and gives some indication of the unobserved burden of disease in the population.
Previous investigations of publicly available sequence data have presented phylogenetic estimates of SARS-CoV-2 time of most recent common ancestor (TMRCA) and growth rates using Bayesian phylogenetic methods [5–7]. Here we use 53 publicly available whole genome sequences sampled up to February 3, 2020 to estimate the growth rate of the epidemic, the size of the epidemic, and the time to most recent common ancestor using a combination of Bayesian and maximum likelihood methods and using a phylodynamic SEIR model.